The monoaminergic system might mediate the link between dysregulation of the tsh receptor and adhd
- Select a language for the TTS:
- UK English Female
- UK English Male
- US English Female
- US English Male
- Australian Female
- Australian Male
- Language selected: (auto detect) - EN
Play all audios:
Access through your institution Buy or subscribe Mice with a knockout of the TSH receptor display behavioural and cognitive features characteristic of attention deficit/hyperactivity
disorder (ADHD). Some of these features were reversed by treatment with methylphenidate, a drug used in ADHD therapy that blocks the dopamine and norepinephrine transporters. Analysis of the
knockout mice revealed monoaminergic changes in several brain regions. This is a preview of subscription content, access via your institution ACCESS OPTIONS Access through your institution
Subscribe to this journal Receive 12 print issues and online access $209.00 per year only $17.42 per issue Learn more Buy this article * Purchase on SpringerLink * Instant access to full
article PDF Buy now Prices may be subject to local taxes which are calculated during checkout ADDITIONAL ACCESS OPTIONS: * Log in * Learn about institutional subscriptions * Read our FAQs *
Contact customer support REFERENCES * Mouri, A. _ et al_. Thyrotoropin receptor knockout changes monoaminergic neuronal system and produces methylphenidate-sensitive emotional and cognitive
dysfunction. _Psychoneuroendocrinology_ 10.1016/j.psyneuen.2014.05.021 Download references RIGHTS AND PERMISSIONS Reprints and permissions ABOUT THIS ARTICLE CITE THIS ARTICLE The
monoaminergic system might mediate the link between dysregulation of the TSH receptor and ADHD. _Nat Rev Endocrinol_ 10, 512 (2014). https://doi.org/10.1038/nrendo.2014.113 Download citation
* Published: 08 July 2014 * Issue Date: September 2014 * DOI: https://doi.org/10.1038/nrendo.2014.113 SHARE THIS ARTICLE Anyone you share the following link with will be able to read this
content: Get shareable link Sorry, a shareable link is not currently available for this article. Copy to clipboard Provided by the Springer Nature SharedIt content-sharing initiative